This surge causes a large amount of testosterone and DHT to be produced in the testes, and thus leads to a tumor growth spurt (termed “tumor flare”). As for agonists, initial application leads to a surge of FSH and LH secretion after their binding to GnRH receptors in gonadotrope cells in the pituitary. ![]() Both agonists and antagonists can suppress Gn secretion and thus block the production of testicular androgens. ![]() Application of GnRH analogs is also known as “ chemical castration”.
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